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Metabolomics analysis To quantitatively analyze metabolic profiles in siNeg HepG2, siHNF4 HepG2, and siNeg SNU449 cells, cells were cultured in regular DMEM + 10% FBS medium
At the molecular level, Tat upregulates miR-34a, which suppresses CARD-containing adaptors and increases NF-B p65, thereby augmenting NF-B signaling and triggering a pro-inflammatory microglial response that contributes to neuroinflammation [111] (Fig
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